Statins Suck.
And why preventing your body from making the most important building block is a bad idea.
If you’ve been reading this Substack or following me on Twitter, you would know that I am not a fan of artificially reducing cholesterol. I am especially not a fan of statins - a class of cholesterol-lowering medication.
When they were first released, statins were hailed as the second coming - an integral part of any risk reduction strategy as it relates to cardiovascular disease.
After the implementation of new penal regulations for clinical trial reporting in 2004, most contemporary data has shed uncertainty on the benefits of taking statins.
This uncertainty has paved the way for research which takes a more critical view of this class of drugs. Its continued failure to meet expectations set by fraudulent pharma-funded research has also provided the population an opportunity to question the cholesterol-basis of heart disease in its entirety.
However, the inertia of the “cholesterol bad” and “statins good” mindset continues to this day.
No matter how much research I present pointing out the physiologically irrational nature of this approach, patients and physicians alike continue to point to statistical sleight-of-hand and treatment apologia.
In this article, I will address these rebuttals and in the process provide an alternative framework for understanding cholesterol and the effects of statins.
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